drugset / Trial / NCT03502577

BCMA-Specific CAR T-Cells Combined With a Gamma Secretase Inhibitor (JSMD194) to Treat Relapsed or Persistent Multiple Myeloma

NCT03502577

NaSingle-groupOpen-labelTreatment

Summary

This phase I trial determines the side effects and best dose of B-cell maturation antigen (BCMA)-chimeric antigen receptor (CAR) T-cells when combined with gamma-secretase inhibitor LY3039478 (JSMD194), cyclophosphamide, and fludarabine in treating participants with multiple myeloma that that has come back or remains despite treatment. Placing genes added in the laboratory into immune T-cells may make the T-cells recognize BCMA, a protein on the surface of cancer cells. JSMD194 may enhance the killing of cancer cells by increasing the BCMA expression on multiple myeloma cells, making the targeted BCMA CAR-T treatment more effective. JSMD194 also decreases the amount of BCMA found in the circulation (called soluble BCMA) that is not bound to the myeloma cells. JSMD194 can therefore reduce the potential for soluble BCMA to act as a decoy. Drugs used in chemotherapy, such as cyclophosphamide and fludarabine, work in different ways to stop the growth of cancer cells, either by killing the cells, by stopping them from dividing, or by stopping them from spreading. Giving BCMA CAR T therapy with JSMD194, cyclophosphamide, and fludarabine may work better in treating participants with relapsed or persistent multiple myeloma.

Timeline

Start
2018-05-23
Primary completion
2022-04-20
Completion
2022-04-20

Drugs

EvaluationDrugModalityDoseRoute
Subject BCMA-specific CAR T-cells Cell therapy 50 cells Intravenous
Subject BCMA-specific CAR T-cells Cell therapy 150 cells Intravenous
Subject BCMA-specific CAR T-cells Cell therapy 300 cells Intravenous
Subject BCMA-specific CAR T-cells Cell therapy 450 cells Intravenous
Subject crenigacestat Small molecule 25 mg Oral
Background Cyclophosphamide Other / unclassified Intravenous
Background Fludarabine Small molecule Intravenous

Indications